They looked at the associations between specific epi-marks and sexual orientation in one group, then tested how well those results could predict sexual orientation in the second group. Actually, Balthazart follows this paradigm. The road to insight should be walked in the opposite direction, potentially offering exciting views. That being so, they miraculously develop a same-sex orientation. It is safe to say that the prenatal history of nearly every homosexual person has been unremarkable from the perspective of endocrinology.
It reminds me of something Noam Chomsky alluded to:
Is Homosexuality Based on a Brain Chemical?
But one of the co-authors, neuroscientist Zhou-Feng Chen of Washington University, cautioned against forming hasty conclusions about the potential influence of this neurotransmitter on human sexual orientation. To state that homosexual subjects were, on average, exposed to atypical endocrine conditions during development, as Balthazart does, seems to me unfounded and biased. This is false because it is our biology that allows us to learn, respond to socialisation, and helps generate our culture. Genes are far from the whole story. In a paper, Rice and his colleagues suggested that such unerased epi-marks might lead to homosexuality when they are passed on from father to daughter or from mother to son.
You can follow LiveScience on Twitter livescience. The road to insight should be walked in the opposite direction, potentially offering exciting views. Order by newest oldest recommendations. In other words, lots of genes which do influence sexual orientation may fall under the radar. In animal experimentation, it is possible to induce in a male animal a largely female sexual repertoire of lordosis and being mounted by a male animal by depriving that animal from exposure to testosterone at the critical period of sexual differentiation of its brain, which comes later in development than the sexual differentiation of the genitalia.